Erdozain et al. (2014) claimed that excessive alcohol consumption can lead to a reduction in brain weight, causing neuronal impairment.
This excessive consumption results in severe damage to enzymes found in the brain that are responsible for the activation of crucial chemical reactions, which contribute to overall brain cell function. But this does not cause cell death.
We see the effects through altered states of behavior and cognition that often result from alcohol interference and deterioration among proteins. If excessive intake persists, the proteins may undergo severe damage, ultimately leading to cognitive impairment.
In the For-Argument, it was stated that neuronal death is permanent; however, in some cases neuronal/cognitive impairment is partly reversible.
Cognitive impairment, due to chronic alcohol abuse, is partly reversible upon abstinence from alcohol consumption (Rainer, M., Mucke, H. M., Chwatal, K., & Havelec, L., 1996 & Tuck, R. R., Brew, B. J., Britton, A. M., & Loewy, J., 1984).
This excessive consumption results in severe damage to enzymes found in the brain that are responsible for the activation of crucial chemical reactions, which contribute to overall brain cell function. But this does not cause cell death.
We see the effects through altered states of behavior and cognition that often result from alcohol interference and deterioration among proteins. If excessive intake persists, the proteins may undergo severe damage, ultimately leading to cognitive impairment.
In the For-Argument, it was stated that neuronal death is permanent; however, in some cases neuronal/cognitive impairment is partly reversible.
Cognitive impairment, due to chronic alcohol abuse, is partly reversible upon abstinence from alcohol consumption (Rainer, M., Mucke, H. M., Chwatal, K., & Havelec, L., 1996 & Tuck, R. R., Brew, B. J., Britton, A. M., & Loewy, J., 1984).
Aziz (2014) studied alcohol-related brain damage and reported on Wernicke-Korsakoff syndrome. This syndrome involves damage to the thalamus, hypothalamus, and the areas of the brain related to memory.
Cognitive impairment results from this widespread damage, but cell death is not seen.
Cognitive impairment results from this widespread damage, but cell death is not seen.